1. Huang C*, et al. Fibroproliferative conditions: the 3R approach bridging fibrosis and tumors. Trends Mol Med. 2025 Apr 22:S1471-4914(25)00060-7.
2. Chen T, Zhang B, Xie H, Huang C*, Wu Q*. GRHL2 regulates keratinocyte EMT-MET dynamics and scar formation during cutaneous wound healing. Cell Death Dis. 2024;15(10):748.
3. Liu L, Yu H, Long Y, You Z, Ogawa R, Du Y*, Huang C*. Asporin inhibits collagen matrix-mediated intercellular mechanocommunications between fibroblasts during keloid progression. FASEB J. 2021;35(7):e21705.
4. Yu H, You Z, Yan X, Liu W, Nan Z, Xing D, Huang C*, Du Y*. TGase-enhanced microtissue assembly in 3D-printed-template-scaffold (3D-MAPS) for large tissue defect reparation. Adv Healthc Mater. 2020;9(18):e2000531.
5. Huang C, et al. Systemic factors that shape cutaneous pathological scarring. FASEB J. 2020;34(10):13171-13184
6. Huang C*, et al. Endothelial dysfunction and mechanobiology in pathological cutaneous scarring: lessons learned from soft tissue fibrosis. Br J Dermatol. 2017;177(5):1248-1255.
7. Huang C, et al. Proapoptotic effect of control-released basic fibroblast growth factor on skin wound healing in a diabetic mouse model. Wound Repair Regen. 2016;24(1):65-74.
8. Huang C, et al. Effect of negative pressure wound therapy on wound healing. Curr Probl Surg. 2014;51(7):301-31.
9. Huang C, et al. Biological effects of cellular stretch on human dermal fibroblasts. J Plast Reconstr Aesthet Surg. 2013;66(12):e351-61.
10. Huang C, et al. Mechanotherapy: revisiting physical therapy and recruiting mechanobiology for a new era in medicine. Trends Mol Med. 2013;19(9):555-64.
